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    A liaF codon deletion abolishes daptomycin bactericidal activity against vancomycin-resistant enterococcus faecalis

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    TY - GEN T1 - A liaF codon deletion abolishes daptomycin bactericidal activity against vancomycin-resistant enterococcus faecalis UR - http://hdl.handle.net/20.500.12495/3555 PB - American Society for Microbiology AB - ER - @misc{20.500.12495_3555, author = {}, title = {A liaF codon deletion abolishes daptomycin bactericidal activity against vancomycin-resistant enterococcus faecalis}, year = {}, abstract = {}, url = {http://hdl.handle.net/20.500.12495/3555} }RT Generic T1 A liaF codon deletion abolishes daptomycin bactericidal activity against vancomycin-resistant enterococcus faecalis LK http://hdl.handle.net/20.500.12495/3555 PB American Society for Microbiology AB OL Spanish (121)
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    Author
    Munita, Jose M.
    Tran, Truc T.
    Diaz, Lorena
    Panesso, Diana 
    Reyes, Jinnethe
    Murray, Barbara E.
    Arias, Cesar A.
    Published in
    Antimicrobial Agents and Chemotherapy, 1098-6596, Vol. 57, No. 6, 2013 p. 2831-2833
    Published for
    American Society for Microbiology
    URI
    http://hdl.handle.net/20.500.12495/3555
    Source's URL
    https://aac.asm.org/content/57/6/2831.long
    DOI
    https://doi.org/10.1128/aac.00021-13

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    Abstract
    The genetic bases for antibiotic tolerance are obscure. Daptomycin (DAP) is a lipopeptide antibiotic with bactericidal activity against enterococci. Using time-kill assays, we provide evidence for the first time that a deletion of isoleucine in position 177 of LiaF, a member of the three-component regulatory system LiaFSR involved in the cell envelope response to antimicrobials, is directly responsible for a DAP-tolerant phenotype and is likely to negatively affect response to DAP therapy.
    Topics
    Enterococcus faecalis
    Vancomicina
    Daptomicina
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